Replacement resorption occurs due to bony remodeling directly follows the resorptive process. If lateral EIRR becomes progressive, it has the potential to progress to replacement resorption, or ankylosis. (Andreasen & Kristerson) When managed in early stages, both apical and lateral EIRR often resolve with non-surgical root canal therapy. (Hammarstrom et al) Transient forms of lateral EIRR called “surface resorption” may occur in the early stages of healing post-trauma, but will resolve as long as pulp tissue retains vitality. These injuries are associated with both pulp necrosis and direct damage to the root surface, including the precementum and adjacent periodontal ligament. (Vier & Figuieredo) Lateral EIRR is often more extensive, and occurs following severe luxation-type injuries or avulsions. Apical EIRR is often present subclinically in cases of apical periodontitis secondary to pulp necrosis. (Tronstad) External inflammatory root resorption (EIRR) relates to endodontic pathosis. (Kaval et al)Įxternal resorptive diseases take several forms depending on their etiology, but share the pathogenesis of loss or damage to the precementum lining the root surface combined with inflammation of the adjacent periodontal ligament, activating odontoclasts. (Caliskan & Turkun) If IRR is perforating, the advent of bioceramic materials as well as regenerative endodontic techniques show promise. In the absence of a perforation, IRR is quite treatable with non-surgical root canal therapy. (Gabor et al) When visible clinically or radiographically, IRR is continuous with the pulp chamber or root canal space. (Wedenberg & Lindskog, Gartner et al) Scanning electron micrographs show that subclinical IRR is found quite frequently in necrotic teeth, indicating that it is likely a part of normal pathophysiology. This combined damage and localized nidus of necrotic pulp tissue incites an inflammatory reaction of the adjacent vital pulp tissue, allowing resorption to progress only until complete pulp necrosis occurs. (Tronstad) It classically occurs following localized coronal pulp necrosis, which may occur secondary to trauma, coronal fractures, deep restorative dentistry without adequate coolant spray, or pulp capping procedures. IRR involves loss or damage to the predentin lining the pulp chamber or root canal spaces combined with inflammation activating odontoclasts. Internal root resorption (IRR) is its own unique entity, whereas external resorption can take many forms. Resorption cannot be simply defined as internal or external.

(Tronstad) The location of this damage, and therefore the associated tissues, determines the type of resorption that occurs. Resorption occurs when developmental precementum or predentin are lost or damaged and inflammation of the adjacent soft tissues allows for clastic cell invasion.

Truly defining resorption requires a much longer answer.īoth resorptive dental diseases and physiologic resorption associated with the exfoliation of primary teeth share a common pathogenesis. (AAE Glossary) This definition really describes what resorption is not. The AAE Glossary of Terms defines Resorption as physiologic or pathologic loss of dentin, cementum, and/or bone not immediately due to caries or trauma. Endo Careers Knowledge Center Submissions.Guidelines for Publishing Papers in the JOE.Oral, Poster & Table Clinic Presentations.

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